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DNA
HEALING HEADLINES
Christianity tells us that God is sending destruction in the form of Armageddon. But just the opposite is happening. From Eta Carina is coming an adjustment of DNA. The Genetic Strand is being changed by laser light being emitted by Eta Carina.
If you would like to see the documentation concerning Eta Carina please CLICK HERE to go to The DNA Page.
CLICK HERE to go to The Genetic Page
FACTS: Eta Carina is the only star in the visible sky that scientists do not understand. The root of the word Protein is Prow which means, The Keel Of A Ship. Protein corrects DNA. The word Carina means Keel Of A Ship. Carina was named for the keel of the ship Argo in the Greek Myth.
The Keel of that ship spoke and gave instructions in time of danger. Carina is fulfilling that prophetic myth and giving instructions to scientists in this magnificent age of Aquarius.
In the mean time, this page will keep you posted on the newest discoveries being made by science relative to healing via DNA. Remember I have said that Cancer, Alzheimers , Aids etc will be eliminated reasonably soon. Nothing can stop the power of Eta Carina, The Seventh Ship with the Keel that gives instructions. It did it in the ancient myth of Jason and the Argonauts and now that myth is being fulfilled.
Please share this page with someone you know who may be suffering from what was previously an incurable disease. Show them this information, and instead of the fear that comes from religion, give them the hope and peace that comes from the cosmic force we call God.
NOW THE DNA HEALING HEADLINES
Brain Cancer Chronic Back Pain
Cancer: Genetically Turning Good Cells Cancerous, Opens A Door
Narcolepsy, Sleep Disorder Sudden Cardiac Death
Cholesterol Gene Genetics and Hemophilia
Genetics and Alzheimers Genetics and Asthma New Drugs
Cancer Bacteria Gene Deciphered
Mouse Diabetes Reversed Skin Cancer Breakthrough
GENE THERAPY MAY IMPROVE BRAIN CANCER TREATMENT
BY PATRICIA REANEY, LONDON (REUTERS)
British scientists are working on new gene therapy techniques to improve brain cancer treatments and to predict how women with breast cancer will respond to treatment.
Researchers at Britain's Institute of Cancer Research and the Royal Marsden Hospital are testing a technique to switch on inactive chemotherapy drugs to kill cancerous cells. So far results of laboratory tests have been encouraging.
"Were seeing substantial cell death," Dr. Gill Ross, one of the leaders of the program, told reporters.
Brain tumors are among the most difficult cancers to treat because many types are resistant to radiotherapy, and chemotherapy drugs have a limited effect on brain tissue.
There are also no screening tests to identify risk factors for the illness, which strikes an estimated 40,000 people in the European union each year.
Nearly half of the patients with brain cancer die within a year of their initial diagnosis.
In the so-called "pro-drug" therapy, the scientists inject bacterial genes, wrapped in a virus, directly into the tumor. The genes trigger an enzyme in the tumor cells which switch on the inactive chemotherapy treatment.
Preliminary laboratory data in tumor types suggest that this pro-drug therapy approach is not only feasible, but also effective, certainly under laboratory conditions said Ross.
MUTANT GENE MAY CAUSE BACK PAIN
THE ASSOCIATED PRESS
NEW YORK (AP) Researchers have identified a mutant gene that may be responsible for a severe kind of back pain caused by ruptured disks in the spine, a finding that could lead to new treatment.
Back pain has long been one of the most poorly understood disorders, though it affects most adults at some point in their lives.
The new research was published in today's Science magazine and reported by The Wall Street Journal.
This study conducted with scientists at the University of Oulu in Finland, analyzed the DNA of 180 Finnish patients with Sciatica. Nine patients were found to have the mutation, and one of the 230 patients in a control group without back pain had it.
The DNA of the extended families of four of the nine patients with the defect revealed a total of 23 relatives with the mutation all of whom suffered disk problems, indicating that the defect likely causes back trouble.
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SCIENTISTS TURN GOOD CELL CANCEROUS
BY DAVID KINNEY, THE ASSOCIATED PRESS
Researchers for the first time have created a cancerous human cell by genetically altering a normal one-an important step toward developing drugs that could one day wipe out cancer.
Scientists already know that cancer is caused by genes turned bad, and they have been trying to develop drugs that fix these flaws.
Until now. they have been fumbling in the dark. They aren't sure exactly which combinations of flaws cause the many types of cancer. Nor do they know precisely which drugs repair which faulty genes.
But biologists at the Massachusetts Institute of Technology's Whitehead Institute for Biomedical Research showed that they can create a cancerous cell with specific genetic flaws in the lab.
The next stop is trying to find drugs that correct those earors.
The work led by MIT's Dr. Robert Weinberg, was reported in today's issue of The Journal Of Nature.
This provides a laboratory tool, and it's a significant one" , said Curt Harris, chief of the National Cancer Institute's Human Carcinogenesis Lab.
Moshe Yaniv, a cancer researcher at France's Pasteur Institute, called Weinbergs work a landmark paper.
By using this latest breakthrough, scientists can work backwards. They can take, say, human breast cells, genetically alter them and see what happens. That way, they can identify some of the major genetic changes that cause breast cancer.
GENETIC FLAW MAY TRIGGER SLEEP DISORDER
THE ASSOCIATED PRESS
BOSTON: Scientists believe they have discovered a genetic flaw deep within the brain that causes narcolepsy, the bizarre disorder that makes people fall asleep without warning.
Two groups of scientists working independently found that narcoleptics overwhelming urge to fall asleep may result from a glitch in signals sent between cells in the hypothalamus, a part of the brain that regulates appetite and other basic drives.
SUDDEN CARDIAC DEATH
HEALTH AND FITNESS NEWS SERVICE
For the first time there seems to be clear evidence that sudden cardiac death in men runs in families.
Although researchers have long known that a person's genetic blueprint contributes to his or her heart attack risk, this is the first study to identify a genetic risk for sudden cardiac death.
CHOLESTEROL GENE
THE ASSOCIATED PRESS
After a four decade search, scientists have identified a gene that regulates the body's level of so called good cholesterol, a breakthrough that could someday lead to a new way to treat one of the most common causes of heart disease.
Flaws in a gene known as ABC1 prevent the production of a protein that the body needs to rinse excess bad cholesterol and other fats out of cells and the blood stream.
GENETICS AND
USA Today: Reporter Tim Friend
Headline: GENETIC DEFECT REPAIRED WITH SYNTHETIC CODE
Technique could permanently correct disorders such as hemophilia in humans. Scientists report they have for the first time permanently repaired a genetic disease in animals with a single drug infusion. They will test the treatment for humans next year. The achievement detailed in today's Proceedings of the National Academy of Sciences holds implications for disorders from inherited single gene defects such as hemophilia, sickle cell anemia and thalassemia (chronic anemia) experts say.
GENETICS AND ALZHEIMERS
Time Magazine:
Adding More Of A Single Gene
The Gene NR2B hisps build a protein called NMDA which acts as a receptor to specific chemical signals. These chemical signals train brain cells to fire in repeating patterns, the patterns are what we experience as memories. A Super Mouse genetically engineered by scientists at Princeton, MIT, and Washington University whose DNA was cleverly altered gives scientists rise to consider that the genetic enhancement of mental and cognitive attributes such as intelligence and memory in mammals is feasible. Scientists have applied to the FDA for permission to do experiments with advanced Alzheimer patients.
GENETICS AND ASTHMA
Reuters News Service:
Reporter Michael Kahn
San Francisco: Scientists have identified two genes that contribute to the development of asthma which could help reduce susceptibility to attacks, researchers said Monday. A new study suggests that just a subtle tweaking of the two newly identified genes could be the key to offering relief to those suffering from the respiratory ailment.
NEW DRUGS
USA Weekend: www.injersey.com
By 2010 we'll have a whole new arsenal of medicines for diseases that are currently untreatable says Carl Feldbaum president of the Biotechnology Industry Organization. Diseases such as Alzheimers and multiple sclerosis will be treated and perhaps reversed by isolated molecules. Patents will be able to rely on the efficacy of new genetically targeted drugs.
CANCER TREATMENT
USA Weekend www.injersey.com
Imagine one of our leading killers reduced to the status of strep throat. Cancer deaths are expected to drop 21% by 2015 with 13% fewer people ever getting cancer. Vaccines that inoculate infants against liver cancer and women against cervical cancer. Drugs will hone in on the cancer via vitamins or drugs engineered to an individuals genetic makeup. Tumors will starve when antiangiogenesis drugs squelch new blood supply to cancer cells.
BACTERIA GENE PATTERN DECIPHERED
Washington AP. Scientists have deciphered the gene pattern of a bacteria that commonly causes meningitis and researchers said it may help in the development of new vaccines.
In a study appearing Friday in the journal , Science, American, British and Italian researchers report they have sequenced the genome, or genetic pattern, of a meningococcus bacterial called Neisseria meningitis, Type B
U.S. TEAM REVERSES MOUSE DIABETES
By Maggie Fox, Health and Science Correspondent
Washington Feb 26 (Reuters). Scientists said on Monday they had used stem cells -"master cells" that are the source of new cells in the body to reverse diabetes in mice.
They said their experiment is a first demonstration that the cells are as valuable as people had said they would be in treating disease. The team at the University of Florida in Gainesville said it has already started testing human cells in the laboratory and think they will work too.
To reverse diabetes you need to take a transplant of the whole pancreas of the islets, Dr Desmond Schatz a professor pediatrics and a diabetes expert at Florida who worked on the experiment said in a telephone interview. Here the potential is you can take stem cells, grow them up , and they grow into islets that are capable of reversing disease.
The next step is to take this into humans, Schatz said. In preliminary experiments it appears that we can take human pancreatic duct cells and show that they can differentiate into islet cells as well.
SKIN CANCER BREAKTHROUGH
LONDON (June 10) -
Scientists have determined that a spontaneous change in a certain gene is
involved in 70 percent of cases of melanoma, the deadliest form of skin cancer,
which kills nearly 40,000 people a year worldwide.
Experts say the finding might lead to more effective drugs for melanoma, which
accounts for just 11 percent of skin cancer, but is hard to treat once it has
spread and accounts for almost all deaths from skin cancer.
Dr. Paul Meltzer, a senior cancer genetics investigator at the U.S. National
Human Genome Research Institute called the finding the biggest breakthrough in
melanoma research for many years.
The discovery, published Sunday in the online version of the journal Nature, is
the first fruit of the Cancer Genome Project, a spin-off of the international
Human Genome Project being run by researchers at the Wellcome Trust Sanger
Institute in Cambridge, England.
Cancer is the disease that lends itself best to an analysis of the human genome
because all cancers are a disease of DNA, said Mike Stratton, head of the Cancer
Genome Project, which aims to identify which of the 30,000 human genes are
involved in cancer and how.
Genes are made up of a DNA code, represented by a sequence of letters. A
mutation occurs when the order of the letters changes.
Mutations can be acquired in two ways: either when DNA is damaged by such toxins
as radiation, chemicals or viruses, or when mistakes are made before cell
division.
Each cell in the body contains a copy of the genome, and duplicates it before it
divides into two. The copy isn't always perfect.
Most of the mutations are harmless. However, sometimes a mutation will occur in
a particular cell in a key gene and the result will be that the gene will be
either switched on or switched off.
That cell will then start to behave abnormally. It will divide when it should
stop dividing. It will move out of its usual position in a tissue and may even
float off into the bloodstream and deposit in another organ.
That is how cancer evolves. Experts estimate it takes about 25 years from the
first gene mutation for a tumor to appear in an adult.
``With the human DNA sequence now available to us, we have started the lengthy
and daunting task of trawling through the vast tracts of genome, gene by gene,
to see if we can find the abnormal genes that drive cells to behave as
cancers,'' said Dr. Andy Futreal, a leader of the Cancer Genome Project.
Meltzer, who was not involved with the research, said the melanoma finding
raises great hopes that the ambitious Cancer Genome Project will pan out.
The scientists start by looking at the genes in 48 tumor samples comprising six
common cancers. They take each one of the 30,000 genes of the human genome in
each sample and look for abnormalities.
After that, they look at the suspect genes in a further 1,000 samples of
cancerous tissue, derived from nearly every type of human cancer, to see how
important a role the gene mutation plays.
The first abnormality they have found is in the gene called B-RAF, which is one
of a chain of genes that must all be switched to the ``on'' position for a cell
to grow and divide.
Normally, it switches on and off, but the scientists found that the mutation
makes the gene stay switched on all the time and ignore prompts to turn itself
off. The cells with the mutation keep multiplying unchecked, leading to cancer.
The researchers found that the code letters in the B-RAF gene were shuffled in
70 percent of melanoma cases, making it the most frequently messed up gene in
melanoma.
The scientists also found that about 10 percent of colon cancers had mutations
in the B-RAF gene and less frequently in a variety of other cancer types.
Stratton said that because the B-RAF mutation was found in 70 percent of
melanoma cases and because the fault in the gene is so specific, it is a
promising target for a new melanoma drug, which would be designed to switch off
the gene only in cells with a mutated version
``We're very excited, but we have to temper that with a certain amount of
caution. Cancers are devious beasts, they are unpredictable beasts. They don't
always respond in the way we would like them to,'' Stratton said. ``We should be
optimistic but we should recognize that the path will take several years.''
Study links six more genes with breast cancer
By Maggie Fox
WASHINGTON, June 13 (Reuters) - Doctors checking into the genetic causes of a
rare children's cancer syndrome said on Thursday they had shown a cluster of six
genes lies behind not only the childhood cancer, but many cases of breast
cancer.
They believe they may have found a complex genetic pathway that causes cancer in
many families, and hope their finding may help identify people most at risk, and
perhaps lead to the development of targeted drugs to treat them.
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