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FORGET AN ARMAGEDDON CHANGE FOR THE WORLD
EMBRACE A DNA HEALING CHANGE FOR THE WORLD
Christianity tells us that God is sending destruction in the form of Armageddon.
But just the opposite is happening.
From Eta Carina is coming an adjustment of DNA.
The Genetic Strand is being changed by laser light being emitted by Eta Carina.
If you would like to see the documentation concerning Eta Carina please :
CLICK HERE to go to The Genetic Page
ETA CARINA AND HEALING DNA
A Brief Explanation Concerning Eta Carina
Eta Carina is the only star in the visible sky that scientists
do not understand.
Carina means The Keel Of A Ship.
Carina was named for the keel of the ship Argo in the Greek Myth.
THE KEEL OF THE SHIP SPOKE
In the Greek myth, it was the Keel Of The Ship Argo, that told Jason
where he could find the Golden Fleece.
Finding the Golden Fleece was important,
because everyone it touched was healed.
THIS MYTHOLOGY SPEAKS TO US NOW
Greek Mythology speaks in a hidden language
about universal cosmic things.
There was no ship Argo. There was no Golden Fleece that healed.
Of course the keel of the ship could not talk. It was wood.
So what was that myth talking about?
NOW IN OUR AGE
THE KEEL OF THE SHIP IS TALKING
The most bizarre star nebula in history is drawing attention
of scientists and astronomers everywhere.
It is the Keel Of The Ship Eta Carina and it is sending energy
to the earth that can impact DNA healing.
The Keel of the ship spoke and gave instructions for healing
in the ancient myth.
Eta Carina is fulfilling that prophetic myth and giving instructions
to scientists in this magnificent age of Aquarius.
If you go to the Hidden Meanings home page,
you will find links to Eta Carina pages so you can be brought up to date
on why I feel that Eta Carina is the Keel of the Ship that will lead
science to the Golden Fleece of DNA.
NOW THE DNA
GENE THERAPY MAY IMPROVE BRAIN CANCER TREATMENT
BY PATRICIA REANEY, LONDON (REUTERS)
British scientists are working on new gene therapy techniques to
improve brain cancer treatments and to predict how women with
breast cancer will respond to treatment.
Researchers at Britain's Institute of Cancer Research and the
Royal Marsden Hospital are testing a technique to switch on inactive
chemotherapy drugs to kill cancerous cells.
So far results of laboratory tests have been encouraging.
"Were seeing substantial cell death,"
Dr. Gill Ross, one of the leaders of the program, told reporters.
Brain tumors are among the most difficult cancers to treat because
many types are resistant to radiotherapy, and chemotherapy drugs
have a limited effect on brain tissue.
There are also no screening tests to identify risk factors for the illness,
which strikes an estimated 40,000 people in the European union each year.
Nearly half of the patients with brain cancer die within a year of their initial diagnosis.
In the so-called "pro-drug" therapy, the scientists inject bacterial genes,
wrapped in a virus, directly into the tumor.
The genes trigger an enzyme in the tumor cells which switch on the inactive
Preliminary laboratory data in tumor types suggest that this
pro-drug therapy approach is not only feasible, but also effective,
certainly under laboratory conditions said Ross.
MUTANT GENE MAY CAUSE BACK PAIN
THE ASSOCIATED PRESS
NEW YORK (AP) Researchers have identified a mutant gene that may be
responsible for a severe kind of back pain caused by ruptured disks in the spine,
a finding that could lead to new treatment.
Back pain has long been one of the most poorly understood disorders,
though it affects most adults at some point in their lives.
The new research was published in today's Science magazine and reported
by The Wall Street Journal.
This study conducted with scientists at the University of Oulu in Finland,
analyzed the DNA of 180 Finnish patients with Sciatica.
Nine patients were found to have the mutation, and one of the 230 patients
in a control group without back pain had it.
The DNA of the extended families of four of the nine patients with
the defect revealed a total of 23 relatives with the mutation all of whom
suffered disk problems, indicating that the defect likely causes back trouble.
SCIENTISTS TURN GOOD CELL CANCEROUS
BY DAVID KINNEY, THE ASSOCIATED PRESS
Researchers for the first time have created a cancerous human cell
by genetically altering a normal one-an important step toward developing drugs
that could one day wipe out cancer.
Scientists already know that cancer is caused by genes turned bad,
and they have been trying to develop drugs that fix these flaws.
Until now. they have been fumbling in the dark.
They aren't sure exactly which combinations of flaws cause the many
types of cancer.
Nor do they know precisely which drugs repair which faulty genes.
But biologists at the Massachusetts Institute of Technology's Whitehead Institute
for Biomedical Research showed that they can create a cancerous cell
with specific genetic flaws in the lab.
The next stop is trying to find drugs that correct those errors.
The work led by MIT's Dr. Robert Weinberg, was reported in today's
issue of The Journal Of Nature.
This provides a laboratory tool, and it's a significant one" , said Curt Harris,
chief of the National Cancer Institute's Human Carcinogenesis Lab.Moshe Yaniv, a cancer researcher at France's Pasteur Institute,
called Weinbergs work a landmark paper.
By using this latest breakthrough, scientists can work backwards.
They can take, say, human breast cells, genetically alter them
and see what happens.
That way, they can identify some of the major genetic changes
that cause breast cancer.
GENETIC FLAW MAY TRIGGER SLEEP DISORDER
THE ASSOCIATED PRESS
BOSTON: Scientists believe they have discovered a genetic flaw deep
within the brain that causes narcolepsy, the bizarre disorder that makes
people fall asleep without warning.
Two groups of scientists working independently found that narcoleptics
overwhelming urge to fall asleep may result from a glitch in signals sent
between cells in the hypothalamus, a part of the brain that
regulates appetite and other basic drives.
SUDDEN CARDIAC DEATH
HEALTH AND FITNESS NEWS SERVICE
For the first time there seems to be clear evidence that
sudden cardiac death in men runs in families.
Although researchers have long known that a person's genetic blueprint
contributes to his or her heart attack risk, this is the first study to
identify a genetic risk for sudden cardiac death.
THE ASSOCIATED PRESS
After a four decade search, scientists have identified a gene
that regulates the body's level of so called good cholesterol,
a breakthrough that could someday lead to a new way to treat one of the
most common causes of heart disease.
Flaws in a gene known as ABC1 prevent the production of a protein
that the body needs to rinse excess bad cholesterol and other fats out
of cells and the blood stream.
GENETICS AND HEMOPHILIA
USA Today: Reporter Tim Friend
Headline: GENETIC DEFECT REPAIRED WITH SYNTHETIC CODE
Technique could permanently correct disorders such as
hemophilia in humans.
Scientists report they have for the first time permanently repaired a
genetic disease in animals with a single drug infusion.
They will test the treatment for humans next year.
The achievement detailed in today's Proceedings of the
National Academy of Sciences holds implications for disorders from i
nherited single gene defects such as hemophilia,
sickle cell anemia and thalassemia (chronic anemia) experts say.
GENETICS AND ALZHEIMERS
Adding More Of A Single Gene
The Gene NR2B hisps build a protein called NMDA which acts as a
receptor to specific chemical signals.
These chemical signals train brain cells to fire in repeating patterns,
the patterns are what we experience as memories.
A Super Mouse genetically engineered by scientists at Princeton,
MIT, and Washington University whose DNA was cleverly altered
gives scientists rise to consider that the genetic enhancement of
mental and cognitive attributes such as intelligence and
memory in mammals is feasible.
Scientists have applied to the FDA for permission to do experiments
with advanced Alzheimer patients.
GENETICS AND ASTHMA
Reuters News Service:
Reporter Michael Kahn
San Francisco: Scientists have identified two genes that contribute
to the development of asthma which could help reduce susceptibility
to attacks, researchers said Monday.
A new study suggests that just a subtle tweaking of the two newly
identified genes could be the key to offering relief to those suffering
from the respiratory ailment.
USA Weekend: www.injersey.com
We'll have a whole new arsenal of medicines for diseases that are
currently untreatable says Carl Feldbaum president of the
Biotechnology Industry Organization.
Diseases such as Alzheimers and multiple sclerosis will be treated and
perhaps reversed by isolated molecules.
Patents will be able to rely on the efficacy of new genetically targeted drugs.
USA Weekend www.injersey.com
Imagine one of our leading killers reduced to the status of strep throat.
Cancer deaths are expected to drop 21% by 2015 with 13% fewer
people ever getting cancer.
Vaccines that inoculate infants against liver cancer, and women against cervical cancer.
Drugs will hone in on the cancer via vitamins or drugs engineered to an individuals
Tumors will starve when antiangiogenesis drugs squelch
new blood supply to cancer cells.
BACTERIA GENE PATTERN DECIPHERED
Washington AP. Scientists have deciphered the gene pattern of a
bacteria that commonly causes meningitis and researchers
said it may help in the development of new vaccines.
In a study appearing Friday in the Journal Of Science, American,
British and Italian researchers report they have sequenced the genome,
or genetic pattern, of a meningococcus bacterial
called Neisseria meningitis, Type B
U.S. TEAM REVERSES MOUSE DIABETES
By Maggie Fox, Health and Science Correspondent
Washington Feb 26 (Reuters). Scientists said on Monday they had
used stem cells -"master cells" that are the source of new cells
in the body to reverse diabetes in mice.
They said their experiment is a first demonstration that the cells are as
valuable as people had said they would be in treating disease.
The team at the University of Florida in Gainesville said it has already
started testing human cells in the laboratory and think they will work too
To reverse diabetes you need to take a transplant of the whole pancreas of the islets,
Dr Desmond Schatz a professor pediatrics and a diabetes expert at Florida who
worked on the experiment said in a telephone interview.
Here the potential is you can take stem cells, grow them up ,
and they grow into islets that are capable of reversing disease.
The next step is to take this into humans, Schatz said.
In preliminary experiments it appears that we can take human pancreatic
duct cells and show that they can differentiate into islet cells as well.
SKIN CANCER BREAKTHROUGH
LONDON (June 10) - Scientists have determined that a spontaneous
change in a certain gene is involved in 70 percent of cases of melanoma,
the deadliest form of skin cancer, which kills nearly 40,000 people a year worldwide.
Experts say the finding might lead to more effective drugs for melanoma,
which accounts for just 11 percent of skin cancer, but is hard to treat once
it has spread and accounts for almost all deaths from skin cancer.
Dr. Paul Meltzer, a senior cancer genetics investigator at the
U.S. National Human Genome Research Institute called the finding the
biggest breakthrough in melanoma research for many years.
The discovery, published Sunday in the online version of the
Journal Of Nature, is the first fruit of the Cancer Genome Project,
a spin-off of the international Human Genome Project being run by
researchers at the Welcome Trust Sanger Institute in Cambridge, England.
Cancer is the disease that lends itself best to an analysis of the human genome
because all cancers are a disease of DNA, said Mike Stratton,
head of the Cancer Genome Project, which aims to identify which of the
30,000 human genes are involved in cancer and how.
Genes are made up of a DNA code, represented by a sequence of letters.
A mutation occurs when
the order of the letters changes.
Mutations can be acquired in two ways: either when DNA is damaged by
such toxins as radiation, chemicals or viruses, or when mistakes are
made before cell division.
Each cell in the body contains a copy of the genome, and duplicates
it before it divides into two.
The copy isn't always perfect.
Most of the mutations are harmless.
However, sometimes a mutation will occur in a particular cell in a key gene
the result will be that the gene will be either switched on or switched off.
That cell will then start to behave abnormally.
It will divide when it should stop dividing.
It will move out of its usual position in a tissue and may even float
off into the bloodstream and deposit in another organ.
That is how cancer evolves.
Experts estimate it takes about 25 years from the first gene mutation
for a tumor to appear in an adult.
``With the human DNA sequence now available to us,
we have started the lengthy and daunting task of trawling through the
vast tracts of genome, gene by gene, to see if we can find the abnormal genes
that drive cells to behave as cancers,''
said Dr. Andy Futreal, a leader of the Cancer Genome Project.
Meltzer, who was not involved with the research, said the melanoma
finding raises great hopes that the ambitious Cancer Genome Project will pan out.
The scientists start by looking at the genes in 48 tumor samples comprising
six common cancers. They take each one of the 30,000 genes of the human
genome in each sample and look for abnormalities.
After that, they look at the suspect genes in a further 1,000 samples of
cancerous tissue, derived from nearly every type of human cancer, to see
how important a role the gene mutation plays.
The first abnormality they have found is in the gene called B-RAF,
which is one of a chain of genes that must all be switched to the
``on'' position for a cell to grow and divide.
Normally, it switches on and off, but the scientists found that the mutation
makes the gene stay switched on all the time and ignore prompts to turn itself off.
The cells with the mutation keep multiplying unchecked, leading to cancer.
The researchers found that the code letters in the B-RAF gene were
shuffled in 70 percent of melanoma cases, making it the most frequently
messed up gene in melanoma.
The scientists also found that about 10 percent of colon cancers had mutations
in the B-RAF gene and less frequently in a variety of other cancer types.
Stratton said that because the B-RAF mutation was found in 70 percent of
melanoma cases and because the fault in the gene is so specific,
it is a promising target for a new melanoma drug, which would be designed
to switch off the gene only in cells with a mutated version
``We're very excited, but we have to temper that with a certain amount of caution.
Cancers are devious beasts, they are unpredictable beasts.
They don't always respond in the way we would like them to,'' Stratton said. `
should be optimistic but we should recognize that the path will take several
Study Links Six More Genes With Breast Cancer
By Maggie Fox
WASHINGTON, June 13 (Reuters) - Doctors checking into the genetic
causes of a rare children's cancer syndrome said on Thursday they had
shown a cluster of six genes lies behind not only the childhood cancer,
but many cases of breast cancer.
They believe they may have found a complex genetic pathway that causes cancer
in many families, and hope their finding may help identify people most at risk,
and perhaps lead to the
development of targeted drugs to treat them.
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Associated Press David Kinney
Bioitechnology Industry Organization Carl Feldbaum
Britain Institute Of Cancer Research
Cancer Genome Project Dr Andy Futreal
Dr Andy Futreal
Images Google unless otherwise noted
Journal of Nature, MIT Dr. Robert Weinberg
Dr. Robert Weinberg
Massachussetts Institute Of Technology Whitehead Institute Biomedical Research
National Academy of Science
National Cancer Institute Curt Harris
National Human Genome Research Institute, Dr Paul Meltzer
Dr Paul Meltzer
Pasteur Institute France Moishe Yaniv
Reuters News Service Michael Kahn
Reuters News Service London Patricia Reaney
Reuters News Service Maggie Fox
Royal Marsden Hospital England Dr. Gil Ross
Dr. Gil Ross
University of Florida Gainsville, Dr Desmond Schatz
Dr Desmond Schatz
University of Oulu in Finland
USA Today Tom Friend
USA Weekend www.injersey.com
Wall Street Journal
Welcome Trust Sanger Institute Cambridge England Mike Stratton